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• View All (505)Cannabidiol provides long-lasting protection against the deleterious effects of inflammation in a viral model of multiple sclerosis: A role for A2A receptors (2013)
This study results highlight the anti-inflammatory effects of cannabidiol in this viral model of multiple sclerosis and demonstrate the significant therapeutic potential of this compound for the treatment of pathologies with an inflammatory component. View study
Cannabidiol Reduces A?-Induced Neuroinflammation and Promotes Hippocampal Neurogenesis through PPAR? Involvement (2011)
Based on the research carried out on rats in this study, it was possible to observe in the results that blocking the peroxisome proliferator-activated receptor-c (PPARc), could significantly reduce the effects of CBD on reactive gliosis and on damage neuronal Also due to its interaction in PPARc, it was observed that CBD stimulates hippocampal neurogenesis. View study
Cannabidiol reduces host immune response and prevents cognitive impairments in Wistar rats submitted to pneumococcal meningitis (2012)
The descriptive results showed that the prolonged treatment with 10 mg / kg of cannabidiol administered to Wistar rats prevented the deterioration of memory in rats with pneumococcal meningitis. Also, it was confirmed that cannabidiol has anti-inflammatory effects in pneumococcal meningitis and prevents cognitive sequelae. View study
Cannabidiol reduces lipopolysaccharide-induced vascular changes and inflammation in the mouse brain: an intravital microscopy study (2011)
CBD exhibits antioxidant and anti-inflammatory properties. These data highlight the anti-inflammatory and vascular stabilizing effects of CBD in endotoxic shock and suggest a possible beneficial effect of this natural cannabinoid. View study
Cannabinoid inhibits HIV-1 Tat-stimulated adhesion of human monocyte-like cells to extracellular matrix proteins (2014)
Based on the results of this study there is evidence that cannabinoids can be used as a therapeutic agent for the eradication of neuroinflammation related to the influx of leukocytes by HIV through the blood-brain barrier (BBB). View study
siRNA knockdown of GPR18 receptors in BV-2 microglia attenuates N-arachidonoyl glycineinduced cell migration (2012)
It is known that neurons use the endogenous cannabinoid system to communicate with other cells of the central nervous system (CNS). Endocannabioid signaling recruits microglia to neurons by activating the cannabinoid CB2 and abnormal receptors of cannabidiol (Abn-CBD). The present study focused on the study of siRNA knockdown of GPR18 receptors in BV-2 microglia attenuates N-arachidonoyl glycineinduced cell migration View study